The smart Trick of apl11 That Nobody is Discussing
The smart Trick of apl11 That Nobody is Discussing
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promyelocytic (M3) acute leukaemia; a number of individuals Categorical an abnormal morphologic spectrum intermediate concerning M2 and M3 AML.
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higher fee of ordinary or dystrophic promyelocytes in peripheralblood As well as in bone marrow; no intracytoplasmic Auer rods; myeloperoxydasereaction beneficial; immunocytochemical detection using an anti-PLZF exhibits adistinct punctate nuclear distribution from the protein, suggesting itscompartmentalization in the nucleus.
Fast detection of prognostically significant fusion transcripts in acute leukemia using simplified multiplex reverse transcription polymerase chain response.
ZBTB16-RARα variant of acute promyelocytic leukemia with tuberculosis: a case report and review of literature.
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Thriving allogeneic bone marrow transplantation in the scenario of variant acute promyelocytic leukemia with ZBTB16-RARA.
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A circumstance of acute promyelocytic leukaemia with abnormal cytological functions as well as a ZBTB16-RARA fusion gene.
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Characterization of cryptic rearrangements and variant translocations in acute promyelocytic leukemia.
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the translocation includes a breakpoint during the zinc fingerregion of PLZF, with fusion of two zinc fingers for the RARa B location toform a five PLZF - three RARa fusion gene; the reciprocal five RARa - three PLZF genefuses 7 zinc fingers to the RARa region; RARas breakpoint happens in1- on account of thealternative splicing of PLZF gene, two types of PLZF-RARa protein is often detected: